Covid19 Part XV - 15,251 in ROI (610 deaths) 2,645 in NI (194 deaths) (19/04) Read OP

eagle eye wrote: »

How do you know we are including all Covid-19 related deaths? With the lack of testing done here I'm sure many have died and not been tested who could have died from Covid-19.

And testing in this country is a complete joke so we are nowhere near knowing the infected figure.

Kermit.de.frog wrote: »

Interesting but this report is more credible source and doesn't deal with notions about labs and all that, rather the cover up.

https://www.youtube.com/watch?v=pEQcvcyzQGE

It's good to see programs like this being aired.

• they were editing sars like corona virus' from bats specifically editing them to include the spike protein to see if they could enter cells in the same way SARs can. (Via ACE2.)
• this person works at the lab in wuhan.
• SARS like corona virus' from bats were engineered to have spike protein for ACE2 reception.

Abstract

Severe acute respiratory syndrome (SARS) is caused by the SARS-associated coronavirus (SARS-CoV), which uses angiotensin-converting enzyme 2 (ACE2) as its receptor for cell entry. A group of SARS-like CoVs (SL-CoVs) has been identified in horseshoe bats. SL-CoVs and SARS-CoVs share identical genome organizations and high sequence identities, with the main exception of the N terminus of the spike protein (S), known to be responsible for receptor binding in CoVs. In this study, we investigated the receptor usage of the SL-CoV S by combining a human immunodeficiency virus-based pseudovirus system with cell lines expressing the ACE2 molecules of human, civet, or horseshoe bat. In addition to full-length S of SL-CoV and SARS-CoV, a series of S chimeras was constructed by inserting different sequences of the SARS-CoV S into the SL-CoV S backbone. Several important observations were made from this study. First, the SL-CoV S was unable to use any of the three ACE2 molecules as its receptor. Second, the SARS-CoV S failed to enter cells expressing the bat ACE2. Third, the chimeric S covering the previously defined receptor-binding domain gained its ability to enter cells via human ACE2, albeit with different efficiencies for different constructs. Fourth, a minimal insert region (amino acids 310 to 518) was found to be sufficient to convert the SL-CoV S from non-ACE2 binding to human ACE2 binding, indicating that the SL-CoV S is largely compatible with SARS-CoV S protein both in structure and in function. The significance of these findings in relation to virus origin, virus recombination, and host switching is discussed.[/I]

spookwoman wrote: »

Sounds too much like those who keep saying the number of deaths are low because most are in nursing homes.
The USA counts nursing homes and they are below us in both % infected and mortality.