Nutritionist/Dietician in Limerick for child with constipation

pprendeville

Looking for a nutritionist/dietician in Limerick area preferably. Our daughter has been suffering from constipation recently and the prescribed laxatives which she has been on for over 2 months don't seem to be doing much. Anyone got any recommendations?

GirlatdRockShow

You can find a list of all qualified dietitians at indi.ie under the find a dietitian tab

2xj3hplqgsbkym

I can't reccommend a dietician but just to let you know that my niece has seen a paediatrician for this problem and she said that no matter how high in fibre her diet may be, this could always be a problem for her and she always has to take the movicol.

joeprivate

Have a read of this from Dr. Greger website nutritionfacts. org/video/childhood-constipation-and-cows-milk/

Transcript: Childhood Constipation and Cow’s Milk the video and more can be seen at the link above
Back in the 50s, it was suggested that some cases of constipation among children might be due to the consumption of cow’s milk, but it wasn't put to the test until 40 years later. We used to think most chronic constipation in infants and young children was all in their head, they were anal retentive, or had some intestinal disorder, but these researchers studied 27 consecutive infants who showed up in their pediatric gastroenterology clinic with chronic idiopathic constipation, meaning they had no idea what was causing it, and tried removing cow milk protein from their diet.

Within three days, 21 out of the 27 children were cured. Symptoms completely regressed when a cow’s milk protein-free diet was used, and there was a clinical relapse during two subsequent cow milk challenges, meaning they then tried to give them back some cow’s milk and the constipation reappeared within 24 to 48 hours. And they did that twice. Same result. They stuck with the milk-free diet, came back a month later, and they stayed cured—and their eczema and wheezing went away too! The researchers concluded that many cases of chronic constipation in young children—more than three quarters it seemed—may be due to an underlying cow’s milk protein allergy.

Chronic constipation is a common problem in children, for which fiber and laxatives are prescribed. If that doesn’t work several laxatives at progressively higher dosages can be used, and that still may not work. Five years later a considerable number of kids are still suffering. In fact it may even extend into adulthood. So to cure the disease in just a few days by eliminating cow’s milk was a real breakthrough.

But this was an open study, meaning not blinded, not placebo-controlled, until … this landmark study was published in the New England Journal of Medicine, a double-blind, crossover study, cow’s milk versus soy milk. Sixty-five kids suffering from chronic constipation, all previously treated with laxatives without success; 49 had anal fissures and inflammation and swelling. An anal fissure is where there’s a rip or tear in the anus, very painful. They gave them either cow’s milk or soy milk for two weeks and then switched it around. So what happened?

In two thirds of the children, constipation resolved while they were receiving soy milk. And the anal fissures and pain were cured, whereas none of the children receiving cow’s milk had a positive response. In the 44 responders, the relation with cow’s milk protein hypersensitivity was confirmed in all cases by a double-blind challenge with cow’s milk. All those lesions, including the most severe anal fissures, disappeared on a cow’s milk-free diet yet reappeared within days after the reintroduction of cow’s milk back into their diet.

This may explain why children drinking more than a cup of milk a day may have eight times the odds of developing an anal fissure. Cutting out cow's milk may help cure anal fissures in adults too, but then give them a cow’s milk challenge and their pain goes from 0 back up to 8 or 9 on a scale of 1 to 10. Cow’s milk may also be a major contributor to recurrent diaper rash.

Why though? All the studies looking at biopsy tissue samples in patients with chronic constipation because of cow’s milk protein hypersensitivity have signs of rectal inflammation. Bottom line, for all children with constipation who do not respond to treatment, a trial of the elimination of cow’s milk should be considered.

Regardless, studies from around the world have subsequently confirmed these findings, curing up to 80% of kids’ constipation by switching to soy milk or rice milk. A common problem with the studies though is when they switched kids from cow’s milk to nondairy milk, the kids could still have been eating other dairy products—they didn’t control the background diet, until now. A 2013 study got constipated kids off all dairy and 100% were cured, compared with the 68% in the New England Journal study where the background diet was unrestricted. In fact in that original study 20 years ago, the cow’s milk was replaced with soy milk or ass milk. Either was better than cow milk, but no mammary milk at all may be best.

To see any graphs, charts, graphics, images, and quotes to which Dr. Greger may be referring, watch the above video

joeprivate

Have a read of this from Dr. Greger website nutritionfacts. org/video/childhood-constipation-and-cows-milk/
Sorry for the long post but it might stop children getting better with some facts

Transcript: Childhood Constipation and Cow’s Milk the video and more can be seen at the link above
Back in the 50s, it was suggested that some cases of constipation among children might be due to the consumption of cow’s milk, but it wasn't put to the test until 40 years later. We used to think most chronic constipation in infants and young children was all in their head, they were anal retentive, or had some intestinal disorder, but these researchers studied 27 consecutive infants who showed up in their pediatric gastroenterology clinic with chronic idiopathic constipation, meaning they had no idea what was causing it, and tried removing cow milk protein from their diet.

Within three days, 21 out of the 27 children were cured. Symptoms completely regressed when a cow’s milk protein-free diet was used, and there was a clinical relapse during two subsequent cow milk challenges, meaning they then tried to give them back some cow’s milk and the constipation reappeared within 24 to 48 hours. And they did that twice. Same result. They stuck with the milk-free diet, came back a month later, and they stayed cured—and their eczema and wheezing went away too! The researchers concluded that many cases of chronic constipation in young children—more than three quarters it seemed—may be due to an underlying cow’s milk protein allergy.




Chronic constipation is a common problem in children, for which fiber and laxatives are prescribed. If that doesn’t work several laxatives at progressively higher dosages can be used, and that still may not work. Five years later a considerable number of kids are still suffering. In fact it may even extend into adulthood. So to cure the disease in just a few days by eliminating cow’s milk was a real breakthrough.

But this was an open study, meaning not blinded, not placebo-controlled, until … this landmark study was published in the New England Journal of Medicine, a double-blind, crossover study, cow’s milk versus soy milk. Sixty-five kids suffering from chronic constipation, all previously treated with laxatives without success; 49 had anal fissures and inflammation and swelling. An anal fissure is where there’s a rip or tear in the anus, very painful. They gave them either cow’s milk or soy milk for two weeks and then switched it around. So what happened?

In two thirds of the children, constipation resolved while they were receiving soy milk. And the anal fissures and pain were cured, whereas none of the children receiving cow’s milk had a positive response. In the 44 responders, the relation with cow’s milk protein hypersensitivity was confirmed in all cases by a double-blind challenge with cow’s milk. All those lesions, including the most severe anal fissures, disappeared on a cow’s milk-free diet yet reappeared within days after the reintroduction of cow’s milk back into their diet.

This may explain why children drinking more than a cup of milk a day may have eight times the odds of developing an anal fissure. Cutting out cow's milk may help cure anal fissures in adults too, but then give them a cow’s milk challenge and their pain goes from 0 back up to 8 or 9 on a scale of 1 to 10. Cow’s milk may also be a major contributor to recurrent diaper rash.

Why though? All the studies looking at biopsy tissue samples in patients with chronic constipation because of cow’s milk protein hypersensitivity have signs of rectal inflammation. Bottom line, for all children with constipation who do not respond to treatment, a trial of the elimination of cow’s milk should be considered.

Regardless, studies from around the world have subsequently confirmed these findings, curing up to 80% of kids’ constipation by switching to soy milk or rice milk. A common problem with the studies though is when they switched kids from cow’s milk to nondairy milk, the kids could still have been eating other dairy products—they didn’t control the background diet, until now. A 2013 study got constipated kids off all dairy and 100% were cured, compared with the 68% in the New England Journal study where the background diet was unrestricted. In fact in that original study 20 years ago, the cow’s milk was replaced with soy milk or ass milk. Either was better than cow milk, but no mammary milk at all may be best.

To see any graphs, charts, graphics, images, and quotes to which Dr. Greger may be referring, watch the above video


Chronic diarrhea is the most common gastrointestinal symptom of intolerance of cow's milk among children. On the basis of a prior open study, we hypothesized that intolerance of cow's milk can also cause severe perianal lesions with pain on defecation and consequent constipation in young children.
We performed a double-blind, crossover study comparing cow's milk with soy milk in 65 children (age range, 11 to 72 months) with chronic constipation (defined as having one bowel movement every 3 to 15 days). All had been referred to a pediatric gastroenterology clinic and had previously been treated with laxatives without success; 49 had anal fissures and perianal erythema or edema. After 15 days of observation, the patients received cow's milk or soy milk for 2 weeks. After a one-week washout period, the feedings were reversed. A response was defined as eight or more bowel movements during a treatment period.
Forty-four of the 65 children (68 percent) had a response while receiving soy milk. Anal fissures and pain with defecation resolved. None of the children who received cow's milk had a response. In all 44 children with a response, the response was confirmed with a double-blind challenge with cow's milk. Children with a response had a higher frequency of coexistent rhinitis, dermatitis, or bronchospasm than those with no response (11 of 44 children vs. 1 of 21, P=0.05); they were also more likely to have anal fissures and erythema or edema at base line (40 of 44 vs. 9 of 21, P<0.001), evidence of inflammation of the rectal mucosa on biopsy (26 of 44 vs. 5 of 21, P=0.008), and signs of hypersensitivity, such as specific IgE antibodies to cow's-milk antigens (31 of 44 vs. 4 of 21, P<0.001).
In young children, chronic constipation can be a manifestation of intolerance of cow's milk.
Article
The causes, prognosis, and treatment of chronic idiopathic constipation in children under the age of six years are still debated.1-4 The two main hypotheses about its causation are that it is psychogenic5,6 and that it results from disturbances in intestinal motility.1,7-9 However, numerous studies have shown that psychological problems are more often the consequence than the cause of constipation.3,4,10-12 The role of alterations in motility still needs to be clarified.13
In an open study of children with chronic constipation, we recently demonstrated that constipation may be a symptom of intolerance of cow's milk.14 We hypothesized that intolerance of cow's milk can cause severe perianal lesions with pain on defecation and consequent constipation and that, in such cases, a diet free of cow's milk can rapidly resolve both the constipation and related disorders. We now report the results of a double-blind, crossover study comparing the effects of cow's milk and soy milk in children with chronic constipation.
All 118 consecutive patients under six years of age with chronic constipation who were referred by family pediatricians to our pediatric gastroenterology clinic between June 1994 and May 1996 were evaluated for study entry. We defined chronic constipation as chronic fecal retention (one bowel movement every 3 to 15 days), often associated with abdominal symptoms (abdominal pain, painful defecation, and so forth). The exclusion criteria were anatomical causes of constipation (Hirschsprung's disease, 1 case; spinal disease, 2 cases), constipation due to another disorder (hypothyroidism, 2 cases; psychomotor retardation, 4 cases), prior anal surgery (2 cases), use of medications that can cause constipation (chlorpromazine, 1 case), and referral for reasons other than chronic constipation (41 cases). Sixty-five children were enrolled (29 boys and 36 girls; age range, 11 to 72 months; mean, 34.6). They were all being fed full-fat cow's milk, dairy products, or commercial formulas derived from cow's milk. Previous treatment with laxatives (mainly lactulose and mineral oil) had been unsuccessful in all 65 patients.
When the patient was first seen, a detailed chart was compiled containing the results of the physical examination and case-history information such as the frequency and duration of breast-feeding during the first months of life, acceptance of formula at weaning, personal and family history of atopic disease, and especially the presence or absence of soiling, abdominal pain, anal fissure, and perianal erythema or edema.
Informed consent was obtained from the parents of all the patients involved in the study, which was approved by the ethics committee of the University Hospital of Palermo.
At the first visit, the parents were asked to record the child's signs and symptoms, and all medications were stopped. Routine laboratory tests and a rectal biopsy were performed. After 15 days of observation, the patient was assigned to receive either soy milk or cow's milk for the next 2 weeks. The milk was supplied in bottles coded A or B by the hospital dispensary. Depending on the patient's dietary habits and age, the total amount of milk given to the patient during the two weeks ranged from 5 to 10 liters. For infants who were less than 15 months of age, the feeding bottles contained a formula based on cow's milk (Transilat, Plasmon, Milan, Italy) or a formula based on soy (Plasmonsoy, Plasmon). For children who were 15 months or older, the bottle contained commercially available whole cow's milk or soy milk. After a one-week washout period, during which the diet was unrestricted, as was the intake of soy or cow's milk and its derivatives, the patients were then switched to the other type of milk for another two weeks. The purpose of the washout period was to cancel the influence of the psychological aspect of the previous treatment period. The order of treatment was randomly assigned by a computer-generated method with the individual patient as the unit of randomization. The researchers were unaware of the order of treatment.
Children with eight or more bowel movements during a treatment period were considered to have had a response. At the end of the two treatment periods, the protocols were evaluated and the code was broken. All children were followed up for a mean of 10 months (range, 3 to 20).
At base line, total serum IgE (with the Phadebas IgE paper radioimmunosorbent test kit, Pharmacia Diagnostics, Uppsala, Sweden) and the erythrocyte sedimentation rate were measured; a circulating eosinophil count and white-cell and red-cell counts were performed, as were the following tests: protein C reaction test, milk-specific IgE antibody assay (Phadebas radioallergosorbent test kit), and skin tests with whole cow's milk, lactalbumin, casein, and β-lactalbumin (Lofarma Diagnostic, Milan, Italy).
The following results were considered elevated or abnormal: total IgE above 60 kallikrein units per liter (the mean value +2 SD recorded in age-matched healthy children by our laboratory); more than 400 eosinophils per cubic millimeter; for milk-specific IgE antibodies, a score of more than 1 (i.e., >4 sorbent units per milliliter); for skin tests, any wheal whose diameter exceeded that of the control and was more than one fourth the diameter of the wheal induced by histamine.
At base line and at the end of the two study periods, the children were examined by a researcher who was unaware of the laboratory test results and the histologic findings. During the study periods, the parents recorded the number of bowel movements, as well as the appearance of the stools and the child's degree of difficulty in passing them according to a previously validated scoring system14 in which a score of 1 indicated mushy or liquid stool, a score of 2 soft feces and no pain on passing stools, and a score of 3 hard feces and difficulty and pain on passing stools.
To ensure that the children did not receive any other kind of milk or milk-containing foods during the study periods, the parents were given a list of the most common milk-containing foods to be avoided. During the study periods, they were asked to record the amount and type of food their child had eaten each day. At the end of the study, we analyzed these diaries to evaluate adherence to the diet and the quantity of milk consumed. The parents were able to contact us whenever necessary, and frequent telephone contacts helped to ensure adherence to the diet.
After the two study periods, the children with a response to the cow's-milk–free diet were given the soy-milk diet for another month and then underwent a double-blind challenge with cow's milk. All the challenges were performed in the hospital, as previously described.14-16 During the challenge, the child was randomly assigned to receive cow's milk or a placebo containing soy milk. The challenge was performed with an initial quantity of 5 ml, and the equivalent of a full feeding (the amount of milk the children customarily received at a meal) was given over a three-hour period. If no clinical reaction was observed within 12 hours after the beginning of the challenge, the patient was discharged and the challenge was continued at home, with bottles coded A or B by the hospital dispensary. During the two-week challenge period, the parents recorded any clinical symptoms, and the patients were reexamined in the hospital for any adverse reaction and at the end of the challenge periods. The challenge was stopped when a clinical reaction occurred — in particular, when there were no bowel movements for 72 hours and the patient had abdominal pain, perianal lesions, or both.
Rectal biopsies were performed in all subjects on the first visit. Twenty patients with a response to the cow's-milk–free diet were selected at random to undergo a second rectal biopsy before the challenge with cow's milk, one month after beginning the cow's-milk–free diet. Biopsy specimens were obtained with Watson's pediatric capsule 4 to 6 cm from the anus. Mucosal specimens were fixed in 10 percent neutral buffered formalin, embedded in paraffin wax, and stained with hematoxylin and eosin, Schiff's periodic acid, and Masson's trichrome. The specimens were evaluated for the following histologic features: abnormal crypt architecture, defined according to Surawicz's criteria17; mixed inflammation (with eosinophils and lymphocytes) of the lamina propria; lymphoid follicles; depletion of goblet-cell mucin; and edema.
Morphometric studies were performed with a Leica interactive image analyzer (model Q500 MC, Leica, Heerbrugg, Switzerland). The following features were assessed: maximal length of surface epithelial cells, the numbers of intraepithelial lymphocytes and eosinophils, and the number of eosinophils in the lamina propria. For the measurement of cellular length, 200 cells per specimen were randomly selected. We counted the numbers of intraepithelial lymphocytes and eosinophils in cross sections of 50 crypts randomly selected from each slide and the numbers of lymphocytes and eosinophils per 100 deep-crypt epithelial cells. The quantitative assessment of eosinophils in the lamina propria was expressed as a percentage of eosinophils per 1000 lamina propria cells per section (five sections per biopsy were examined). The pathologist examining the biopsy specimens was unaware of the subjects' laboratory test results and the clinical response to the diet.
Frequency analysis was performed with Fisher's exact test. The Wilcoxon rank–sum test was used to compare the number of bowel movements per day and the qualitative fecal scores. Student's t-test (for parametric analysis) or the Mann–Whitney U test (for nonparametric analysis) was used to compare the morphometric data on the rectal mucosa. All P values were two-tailed, and P values of less than 0.05 were considered to indicate statistical significance.18
The base-line characteristics of the 65 patients are shown in Table 1TABLE 1
Base-Line Characteristics of the 65 Patients.
. During the first study period, 33 patients received cow's milk and 32 soy milk; 21 of the children who received soy milk and none of the children who received cow's milk had a response (P<0.001). During the second study period, 32 children received cow's milk and 33 soy milk; 23 of the children who received soy milk and none of the children who received cow's milk had a response (P<0.001). Table 2TABLE 2
Number of Bowel Movements and Qualitative Fecal Scores during Each Study Period for the 65 Patients.
shows the number of bowel movements and the qualitative fecal scores during the two study periods. The number of bowel movements significantly increased and the fecal score significantly improved when the patients were receiving the soy diet.
Forty-four of the 65 patients (68 percent) had a response to the soy-milk diet. During this period, all those with a response had at least one bowel movement daily after two to six days, soft feces, and no discomfort on defecation, and erythema and perianal edema completely resolved. Six patients did not complete the protocol. All six had had a response to the soy diet during the first study period, but 9 to 12 days after starting the cow's-milk diet, they again began to have constipation and were withdrawn from the study. These six were included in the intention-to-treat analysis.
All 44 children with a response to the cow's-milk–free diet underwent a double-blind, placebo-controlled challenge with cow's milk in the hospital after a further month on the diet. None of those who received the placebo (soy milk) had a clinical reaction. No patient who received cow's milk had an acute reaction, but in all patients constipation associated with hard stools and discomfort on defecation reappeared after 5 to 10 days of the diet. The cow's-milk–free diet was therefore recommenced, with a consequent normalization of bowel movements in all patients. Thus, 44 of the 65 patients were given a diagnosis of constipation related to intolerance of cow's milk.
The severity of constipation in children with a response and in those without a response was similar at base line. However, anal fissures with erythema or edema were more common among those with a response (40 of 44 patients vs. 9 of 21, P<0.001). Anal lesions tended to disappear within the first seven days after the initiation of the soy-milk diet and reappeared three to six days after the reintroduction of cow's milk, often before the reappearance of constipation. Furthermore, at diagnosis, symptoms of suspected intolerance of cow's milk were more common in children with a response (11 of 44 patients vs. 1 of 21, P=0.05): recurrent bronchospasm in 4 patients, rhinitis in 4, and dermatitis in 3.
At entry into the study, there was a trend toward a higher frequency of positive skin tests among the patients with a response (10 of 44 vs. 1 of 21, P=0.07) and a higher frequency of specific IgE antibodies to cow's milk antigens (18 of 44 vs. 2 of 21, P=0.009). At entry, 31 of the children with a response had positive results for one or more of the immunologic tests, as compared with 4 of the children with no response (P<0.001). During the follow-up, after four to eight months, there was a normalization or a significant reduction in serum immunologic values (data not shown) in the children with a response.
None of the patients had alterations in liver and kidney function or in indicators of metabolism or inflammation (white-cell count or erythrocyte sedimentation rate and protein C reaction) during the study.
Table 3TABLE 3
Histologic and Morphometric Findings According to the Response to a Cow's-Milk–free Diet.
shows the histologic findings of the rectal biopsies. The length of surface epithelial cells was significantly shorter in the children with a response than in those with no response, giving a cuboidal aspect to the surface epithelium. Furthermore, inflammation was more frequent in the children with a response; the inflammation was characterized by infiltration by lymphocytes, but the most relevant finding was the infiltration of the lamina propria by eosinophils and the presence of intraepithelial eosinophils in the crypts (Figure 1AFIGURE 1
Rectal Mucosa in a Patient with Constipation Related to Intolerance of Cow's Milk at Base Line, Showing Infiltration of the Mucosa by Eosinophils.
and Figure 1B). We found inflammatory changes in 26 of the 44 children with a response and in 5 of the 21 children with no response (P=0.008). One or more histologic alterations were found in the rectal mucosa in all 44 children with a response and in 12 of the children with no response (P<0.001). A random sample of 20 children with a response underwent a second rectal biopsy immediately before they underwent the challenge (one month after they began the cow's-milk–free diet). The histologic findings were normal in 8 patients and considerably improved in the other 12 patients (reduced inflammation, disappearance of lymphoid nodules, reduction in the cuboidal aspect of the surface epithelium, and increased mucin in the glandular cells).
The mean (±SD) daily consumption was 450±120 ml of soy milk and 470±135 ml of cow's milk. Analysis of the main constituents of the diet (proteins, carbohydrates, and fibers) did not show any qualitative or quantitative variations during the study period (data not shown).
During the follow-up, none of the children with a response had constipation; cow's milk was reintroduced into the diets of 15 children after 8 to 12 months of the cow's-milk–free diet, and in all cases constipation returned within 5 to 10 days. The children with no response to the soy-milk diet were treated with high doses of laxatives, with a subsequent improvement in stool frequency. In all cases, however, the symptoms returned once treatment with laxatives was stopped.
Although chronic diarrhea is the most common gastrointestinal symptom of intolerance of cow's milk in children, our current results confirm our earlier observation that chronic constipation can also occur.14 We found a relation between the intake of cow's milk and constipation in about two thirds of the patients studied. However, it is possible that the children noticed the difference in taste between cow's milk and soy milk. Thus, we cannot totally rule out psychological factors, which are frequently suggested as the cause of chronic constipation.5,6 On the other hand, we did not find any evidence of a psychological aversion to cow's milk, such as a refusal of the formula or prolongation of breast-feeding in infants being weaned onto formula containing cow's milk. Furthermore, the results of the double-blind challenge with cow's milk confirmed that constipation was a symptom of intolerance of cow's milk in most of our patients.
We may have overestimated the frequency of intolerance of cow's milk as a cause of constipation. Our gastroenterology center has experience in the treatment of food allergies, and the pediatricians who referred patients to our clinic may thus have preselected our patients. Furthermore, all the patients had been previously treated unsuccessfully with laxatives; thus, the high frequency of constipation related to intolerance of cow's milk in our study may have been due in part to our inclusion of patients with no response to laxatives. On the basis of these considerations, we must emphasize the highly selected nature of our group of patients.
Immunologic tests showed hypersensitivity in nearly three quarters of the children with constipation related to intolerance of cow's milk, with the most common mechanisms being IgE-mediated; thus, hypersensitivity increases the probability that constipation will be a manifestation of food intolerance. Although the rectal biopsy did not show any specific mucosal alterations, signs of inflammation were much more frequent in the children with a response. The infiltration of eosinophils is the main characteristic of patients with intolerance of cow's milk, but as has been reported previously,19,20 we often observed a mixture of lymphocytes, eosinophils, and monocytes.
Our data suggest that the concomitant presence of other manifestations of intolerance of cow's milk (bronchospasm, dermatitis, and rhinitis) increases the probability that constipation will be found to be a symptom of intolerance of cow's milk. However, as others have reported, constipation may be the only presenting symptom.21,22 Furthermore, clinical examination of the children in our study showed a very high frequency of severe anal fissures. Because these lesions reappeared after the reintroduction of cow's milk and before the onset of constipation, we hypothesize that they are one of the mechanisms causing constipation. Pain on defecation can cause retention of feces in the rectum, with consequent dehydration and hardening of the stools, thus aggravating constipation.

Alf Veedersane

You could just link it and reduce the visual assault from the wall of text you've copied and pasted.

Or just not.

joeprivate

My settings wont allow me to post links

Alf Veedersane

joeprivate wrote: »

My settings wont allow me to post links

Modify the link and post it. Leave a space before the .com so it's not a direct link but people can copy, amend and go to if they want to.

pprendeville

Git it, if you want you can edit that thread and delete the majority of the text. It's some mouthful alright.

joeprivate

Folk
I understand where your coming for ,its a long post, but I hoped that the message that milk can cause constipation in young children had a better chance of been read if I posted the text rather than a broken link that not everybody understands how to fix.

mansize

Posting it twice is super fcuking annoying

joeprivate

Sorry did not mean to annoy anybody and definitely did not mean to "super fcuking annoying" anybody .
I tryed to edit the first post to add a extra bit to it but ended up posting part of the post twice and at this point I still cant edit it perhaps because it so long.
But on the other hand it it stops just one child some where suffering with constipation your "super fcuking annoying" feelings will be balanced out by the a child who is cured from this condition cause I am sure that pain is almost could fall into the "super fcuking annoying" category.
If I can figure out how to edit the first post I will but at this point the boards software seems to freeze when I try to edit it.

mansize

Try the MMR vaccine

Alf Veedersane

joeprivate wrote: »

Folk
I understand where your coming for ,its a long post, but I hoped that the message that milk can cause constipation in young children had a better chance of been read if I posted the text rather than a broken link that not everybody understands how to fix.

If someone is interested they'll try the link. It helps if you explain what you've done as well. People are less likely to try scale a wall of text.

As for the thrust of the study, there are more likely causes than milk.